BLU board member Jared Lloyd was recently interviewed for a Sheep Industry News article. The article focuses on his flock expansion using Shetland and BFL genetics in a large western commercial flock. http://sheepindustrynews.org/?page=site%2Ftext&nav_id=3b79b0fae814183646395f41f85dc25e&archive_id
Contributed by Margaret Fryatt, British Columbia, Canada
On June 4th, 2011, Vancouver Island Fleece Judge, Matt Robley, evaluated fleeces entered by local sheep breeders. Of the 51 fleeces entered in the show, most came from the Fraser Valley. There were entries from further afield but my three entries traveled the greatest distance to be included. Fleeces were sifted by a committee of 22 association members prior to the show. Only those meeting the standards set by the LMPSA were judged. This event was hosted at the Belmont Farm, one of Langley’s heritage dairy farms. The facilities were perfect for the event with lots of room to spread out the fleeces for judging and also space for local vendors to ply their wares. After judging which was open to the public, fleeces were placed on display.
Just prior to the sale itself, the awards were announced. I was quite delighted to learn that one of my fleeces had been awarded the Glibbery/Tuytel Spinners Delight award. What could be more fitting than this! The award was started by the LMSPA to encourage producers to produce fleece that would encourage a spinner and recognize the great local fleeces available. The name of the award comes from two very prominent sheep raising families that have been very active and long time members in the LMSPA. The award does not necessarily go to the fleece with the highest score, but is selected by the judge as the one fleece that he or she deems to be the fleece that would most appeal to the spinning enthusiast best representing the qualities of hand, softness, staple length and luster. It was very exciting to have a Bluefaced Leicester fleece selected.
Many spinners in BC have not had the opportunity to see a BFL fleece in the grease and so there was always quite a crowd gathered around the tables. Many cries of delight were heard and I understand that the competition to purchase fleeces was very strong. I think that the fleeces were lovely and well represented the qualities of BFL fibre.
Many people are still unfamiliar with Bluefaced Leicester fiber and it is always fun to show them how soft and lustrous it is. It is my very favorite fibre to spin with and I find that more and more folks agree with me. Once you are hooked, you are hooked for the long term.
Of special significance for me is the fact that the award is named after one of the Shuswap Spinners and Weavers Guild members, Judith Glibbery. She and her husband made a huge contribution to sheep and fleece production during their tenure in the Lower Mainland and dedicating the award to them was intended to recognize their involvement. It certainly made winning this particular award extra meaningful for me!
From BLU member Nancy Gilkeson
Well I want to share a shocking event with you all! My daughter Christie talked me into taking two BFL ewe lambs to show in open class at the Douglas County (Oregon) fair which is going on now. It was the first time ever for me to show at fair and only 2nd time ever to show my sheep. I have enjoyed that process by the granddaughters for a handful of years.
Our county has quite a few sheep but primarily the meat breeds who show there. Very few wool or dual use breeds are shown and so these all end up in “other” category. We showed along side the Shetlands.
My oldest BFL ewe lamb was nice and won first in the class with the younger lamb in third place out of five. Not much verification for me that “Jenny” is indeed wonderful but of course nice to get blue and Champion ribbons on her.
The thriller came when all champion ewes and ewe lambs from all the other classes (Suffolk, Dorset, Horned Dorset, St Croix, Cheviot, North Country Cheviot, Southdown, etc) were in the ring and the judge chose our BFL ewe lamb as Grand Champion Ewe in Open Class!!! Christie had said not to expect much with that Champion round as the top sheep in meat breeds are tough to compete against.
I could not be more thrilled with this win!!!
By Kathleen Davidson
To a shepherd, homegrown fiber is priceless so sending it off to be processed can be stressful, to say the least. All the effort in growing and harvesting our wool should be reflected in the perfect processed product. But sometimes the returned product doesn’t meet our expectations.
What went wrong? Hopefully I give give some helpful hints to prevent surprise or catastrophe with your fiber. Lessons I learned by mistakes in my 25 years of fiber processing.
LESSON 1: Be very specific about what you want and label all the bags of wool in the box. One time I sent 5 batches of dyed wool for roving expecting 5 batches of roving to be returned. To my surprise, all 5 batches had been carded into one big technicolor cloud. I thought I had labeled everything clearly enough but I guess there was ambiguity somewhere.
LESSON 2: Even thought it costs more,separate boxes could be a good idea. Years ago I stuffed my precious Targhee fleeces that I had accumulated for 3 years in a box with Romney fleeces. Yup, they were carded together even though my directions clearly said, make Targhee roving and Romney roving.
Don’t assume the processor knows breed characteristics in fiber. Label, label, label.
LESSON 3: To wash or not to wash before shipping. You will save both shipping and processing money sending washed fiber if you can get it done at home but don’t be surprised if your fiber is rewashed at the mill because it was “sticky”. Mills value their expensive equipment and sticky fleeces can do damage.
Test your washed fiber by letting it sit in the sun for a day. If it feels sticky it will need to be washed again. Rewashing at the mill is sometimes a surprise expense but not a bad thing for the final product.
LESSON 4: Pick through your fleeces, removing as much vegetation as possible. A lot of vegetation falls out in picking and carding but if there is too much it will show up in your yarn. Check fleeces for weak tips and breaks.
It hurts to toss a fleece from your favorite ewe but sometimes you have to bite the bullet and chalk this year’s fleece up as a loss if it has weakness that will ruin a yarn run.
LESSON 5: Check the mill’s minimum weight requirements. These vary. Make sure you send enough fiber to meet the requirements. Calculate for loss in washing and carding. I usually send 32 pounds of greasy wool to make 15 pounds of yarn.
Some mills will call you if you need to send more fiber but others will just charge the minimum rate and surprise you with fewer skeins and a bigger bill. There are mills with no minimum weights for processing if you only want a few fleeces turned into yarn.
LESSON 6: Talk to the folks at the mill and tell them how you want your yarn to finish. Discuss weight, yards per skein, ply and whether you want it in cones or skeins. Don’t be surprised if you get a different weight than you had hoped for. Be flexible.
One mill I use lets the wool “tell her how it wants to be spun”. Another mill surprised me with the most gorgeous bouncy BFL yarn I have ever felt. The people that run the mills know the process much better than I do so I give them creative power with my fiber. I have never been disappointed.
The best thing to do is look at the yarn of other breeders. Buy a skein or two and knit with it. When you find the yarn that works for you ask who their processor is. Some breeders sell soft lofty skein while others have a more crisp yarn for definition of cables and Aran patterns.
It’s all what you like and want your yarn to be. I use 3 different mills and although they spin different yarns for me, I would recommend all of them. Ask me about my yarn anytime!
By Robina Koenig, Tumble Creek Farm
As one crop of lambs arrives, I plan next year’s breedings and ponder this very topic. Should I use AI again this fall or wait until next year? How good are my current rams? These and other questions pile up as I make preliminary decisions for my flock.
There are several things to consider regarding the use of LAI (laparoscopic artificial insemination). The following questions outline my view, and are the framework for my breeding-season decisions.
Does AI fit into my long-range goals for my flock? I was fortunate to have a friend share her thoughts on goal-setting when I first acquired sheep. There are immediate goals—short term of 2 to 5 years— then the long term of 10 or more years.
Of course these will change and be updated as time passes but establishing goals has helped me keep focused. The obvious goal in using AI is genetic progress at a greatly accelerated rate.
Can I afford it? What will it do to my budget? Who/what/where is my market, and how many ewes do I want to commit to? In the choice to use AI, there’s the cost of semen, hormone synchronization supplies, and the technician’s fees to count. There’s my own extra work in preparing my ewes and facility.
If AI will be done at my farm, I will need special equipment available for the technician plus a crew of helpers; if it will be done at someone else’s farm, I need to consider transport, perhaps for many miles. My own accomodations would need to be arranged as well if it is too late to drive home when AI is finished.
Which ram(s) do I want to use? This query takes me the most time to answer. Studying the rams’ pictures and pedigrees for traits and characteristics I’d like to add to my flock is the basis for this decision. A picture may be worth a thousand words, but it can be difficult to evaluate any animal from a single photograph which may or may not have been taken under ideal circumstances. Comments from other breeders who have used the ram, the AI technician, and perhaps the ram’s owner are very helpful.
When shall I schedule AI? When do I want the lambs born? I answer this based on how big I want my lambs for the first show I attend each year, which is the Black Sheep Gathering (BSG), in late June.
Working backward to select an AI date usually runs me into the last show I attend, Oregon Flock and Fiber Festival (OFFF). One of the benefits of AI is the small window of time for lambing—four or five days—of the ewes that conceived to AI. Timing is everything!
What about bringing in a ram (perhaps an AI offspring) from another flock? The old saying “A ram is half your flock” justifies considerable cost, but that expense may still be less than AI, and it eliminates the investment in time and money involved in the AI preparation and procedure.
Using a first-generation AI offspring ram will carry many benefits to my future lamb crop from very remote and diverse sources. Of course the lambing date widens a bit in pasture breeding if I need to plan lambing time around another event on my calendar.
The questions and answers above will change somewhat if I have my own collection of rams from previous AI breedings. The obvious one is cost, but I seriously consider his pedigree and the less predictable lambing dates.
If I do use my own ram then I’ve seen how he’s grown in body and fleece to evaluate his potential. To me, this is what really demonstrates the benefits of AI genetics for the future. Remarkable improvements in all the traits we value as sheep producers can be obtained using AI or AI offspring in our breeding programs.
When I first considered AI there was only one Bluefaced Leicester sire available, Gwestydd Jamie. Working through the new learning curve of hormone therapy, I took my four ewes 150 miles to team up with two friends for a cold January AI session. One of the four ewes settled and produced two ewe lambs. I was ecstatic!
Rates of conception to AI since then have been quite high; several years 100% of my ewes took to AI with excellent results including a set of quadruplets.
The picture is still changing with new rams that have been selected for import. Choose wisely, my friends.
Three diseases that have become frighteningly prevalent in the US sheep flock are little understood by the average shepherd. All three of these diseases are slow to show symptoms, are contagious before they are detected, and will significantly shorten the life of your ewes and otherwise lower their productivity.
CL is contagious to humans and there is some suspicions Johnes may be associated with Crohns disease (although there is more risk of Johnes from the cow’s milk in your refrigerator than your sheep). Given the prevalence of all three and the myriad myths surrounding them, taking some time to learn the facts can save you plenty of trouble.
The good news is that there are now excellent tests for all three diseases that are conclusive and not difficult to find a lab to do. Due to some incidents of false positives when a sheep shows positive for the disease on the OPP ELISA test, a different immunological test can be used to back up whether the sheep is really positive. There are a variety of tests for Johnes, depending on whether the feces, blood, or milk is being tested.
The Synergistic Hemolysin Inhibition (SHI) Test is used for CL and results are graded on intensity – pick an experienced lab for this one. The University of California, Davis lab has a high degree of proficiency in CL testing. Many state labs do both Johnes and OPP. Frequently, testing is subsidized by the state, so if you test in your own state your testing costs will be significantly less than if you use an out of state lab.
The bad news is that there are no cures and no really effective vaccines available for any of these diseases. Testing and removing positive animals as soon as they are identified is the only effective way to eliminate these diseases from a herd.
All three of these diseases have been found in the BFL herd in the US. It helps no-one to point fingers or keep the disease status of their animals secret. These diseases are too prevalent in the US to be sure that any herd that is not 100% closed has not acquired at least one of these infections.
All three diseases can be spread at shows. They come into your flock with perfectly healthy looking stock that did fine in quarantine. Your neighbors’ animals, their boots, and their equipment can be vectors if their flocks are infected. That dandy stud ram you borrowed can be carrying one of these diseases. What will make a difference is honest cooperation to minimize any potential risk of transfer and willingness to test our herds and the sheep that we sell.
Some sheep are more susceptible than others. Some can carry one of the diseases without ever being affected by it; some will be chronically afflicted by it very quickly. But, once exposed to the bacteria (including simply being with another sheep that has been exposed), a sheep must be considered infected unless testing proves they are not. All three diseases have been studied extensively and it is clear they all create a drag on the animals system, resulting in shorter lives and greater susceptibility to other problems and diseases in most infected individuals.
It is possible to continue to obtain clean lambs from valuable but infected breeding ewes if they are still reasonably healthy. In order to prevent infection of the lambs, the lambs need to be removed at birth, allowing as little contact with the mother as possible. She shouldn’t lick them. The lambs must be prevented from drinking any of the infected ewe’s milk or colostrums. In the case of Johnes, no feces contact is critical.
Ewes with CL should be examined carefully before they give birth and any CL cysts treated with antibiotic and cleaned out in order to minimize the possibility of a cyst contaminating the lamb. Any ewe with a chronic cough should be monitored very carefully to be sure she doesn’t contact her lamb.
Studies have shown that the likelihood of the disease agents crossing the placenta and infecting the lamb prior to birth are next to none, unless the ewe is very ill and her immune system has started to shut down. In any case, the lambs shouldn’t be introduced to the clean flock until they are tested at 6 months of age. By then any passive antibodies they have received from their mothers would be gone and a positive test would indicate active infection. While they still have antibodies from their mother, they may test positive, but not be infected.
The OPP society website has excellent directions and advice for anyone wanting to raise lambs that have been removed from their mother at birth. They have directions for pasteurizing colostrum and methods of feeding groups of lambs to get them off to a good start. Remember that cow colostrum often carries Johnes, so don’t use it without pasteurizing.
Caseous Lymphitis (CL or Cheesy Gland) is caused by a tuberculosis type bacteria that lives inside of cells, colonizes the sheep’s lymph system, and forms cysts in the lymph nodes that protect the bacteria from antibiotic. The disease is also common in goats. The bacteria is passed when cysts close to the skin, in the lungs, or in the mammary glands burst releasing thousands of bacteria into the environment and the bacteria is rubbed on skin, inhaled by other sheep, or passed through milk to the lambs.
The bacteria can live over a year in the environment and is quite good at entering through the skin of the sheep, even in the absence of a wound. Initial skin cysts may not be noticed and internal cysts cannot be seen. The animals system is weakened by the disease and production is seriously affected. Most animals are shipped before the CL becomes advanced, due to poor production.
The first cyst is typically confined to skin, but the bacteria move from the initial infection into the lymph system, where they encyst and hide from any antibiotic treatment and the animal’s immune system. CL will eventually take over the lymph system and the animal becomes emaciated and covered with oozing cysts. Many countries condemn the meat of animals when cysts are observed during slaughter. The US does not, but requires cysts in the meat to be cut out.
CL can infect humans and treatment is painful, expensive, and takes years. Sheep shearers are at risk because shearing commonly bursts any cysts present at the surface. The dipping bath post shearing has been identified as a major avenue of transmission for sheep in Australia and New Zealand. Injections cause a break in the skin that is another easy route of entry for the bacteria. Don’t use the same needle on different sheep and use an antiseptic to sterilize the skin at the site of the injection.
There is an inoculation called Colorado serum for CL, but it is generally considered only about 75% effective in preventing infection, it does not prevent cyst formation in already infected ewes, and it seems to cause some severe adverse effects in ewes that have already been infected. The biggest problem it poses for a purebred herd is that once an animal is inoculated it is impossible to tell if they have an active infection or if the antibodies in their blood are only due to the inoculation. The inoculation has been useful in reducing the economic impact of CL in commercial herds.
If you do decide to manage CL in an infected herd kept for meat production or in quarantined ewes being kept for breeding, there are some newer penicillins and tetracyclines in carriers that penetrate the puss in the cysts better. Cysts are best cleaned up by injecting them with one of these antibiotics well before they start to burst. Once the bacteria in them are dead, they dry up and are much less of a risk to other sheep. Talk to your vet about the best antibiotic to use.
Shearers , mostly in Australia and New Zealand are known to catch CL. They are probably exposed frequently through cysts that burst when they shear. There are other infrequent examples of people contracting the disease. Anyone cleaning and treating a cyst should be very careful not to expose their skin to the bacteria if they suspect CL. The disease is very expensive and takes a long time to eliminate in humans, frequently requiring surgery to eliminate infected lymph nodes.
The incident of CL in sheep is growing and has gone from a minor problem in the US to a major one in the last 10-15 years. Many commercial flocks are infected through new rams brought from purebred flocks. This is an unacceptable situation and it is paramount that purebred breeders behave responsibly to prevent the spread of the disease.
In Australia, neglect of the disease created such a problem that now vaccination of sheep is required by law and movement of animals from known infected flocks is restricted (they have a better vaccine in Australia). Unfortunately, many sheep farmers in the US feel that since CL is a slowly progressing disease, they do not need to remove a ewe until she becomes unthrifty and they have ‘gotten their money out of her.’
John Glenn, DVM at UC Davis has published some articles that clearly explain your options. A description of the tests can be found at this link for www.goatworld.
Johnes disease is found in sheep, goats, and cattle. It is another tuberculin type bacteria that infects the intestinal wall cells and becomes systemic as the disease advances into late stages. It slowly destroys the ability of the intestine to absorb nutrients. Poor doers, due to this disease, typically start to show low condition in spite of a good appetite during the second to third year and ewes commonly need culling at age 3-4.
As the disease progresses, the animal will starve to death while demonstrating a healthy appetite. The bacteria is present in the milk of an infected animal and infection of nursing young is almost 100% in calves. Little investigation has been done on sheep, but the organism has been found in sheep milk and is likely just as infection to lambs. Lambs will also contract the disease from cow colostrum.
The disease is endemic in US dairy cows and programs to address it in cattle have been started in states where dairy is an important part of the economy. Johnes bacteria have been linked to Crohn’s disease in humans, but there has been no conclusive evidence yet that it causes the disease.
In cattle and sheep, the bacteria is passed from one adult animal to the next through fecal contamination and ingesting the bacteria. The bacteria are shed from the intestinal wall during all stages of infection. Sometimes in early infection, a blood ELISA test will not be positive, but fecal testing will show the presence of the bacteria. Feeding off the ground and frequently moving pasture is recommended to minimize contamination.
Both blood ELISA testing and fecal testing are used for Johnes. If you want to find out if Johnes is an issue with your flock, collecting a representative sample of your flocks droppings and having the mixed group tested is a low cost approach to finding out if you need to investigate further. There is also a milk test used for dairy cattle, but most sheep producers will want to know if their animals are sick before they produce lambs.
There is a vaccine for Johnes for cattle, but it isn’t used in sheep. It has nasty side effects in both species and causes serious illness in humans if they are accidentally dosed with it. There is a new vaccine for sheep developed by Cornell University, but it has not been approved yet and is not on the market.
Johnes is less common in sheep than OPP and CL, but the problem is growing and if ignored will only get worse. It is a huge problem in the US dairy herd and major testing programs have begun in states where dairy is important. Check out the Wisconsin Johnes website for more resources at www.johnes.org
Ovine Progressive Pneumonia (OPP or Maedi Visna) is a retrovirus disease similar to AIDs in humans. It is does not infect humans. It causes a wasting disease in sheep, the common symptoms being hardened and unproductive mammary glands and chronic pneumonia that will not respond to antibiotics. The virus slowly destroys the immune system in the sheep, just like AIDs does in humans.
In advanced cases, neurological damage causing trembling and uncontrolled spasming of muscles may be seen. The disease is usually passed through breathing the virus in aerosol from a coughing sheep or to lambs through their mother’s milk. Studies shoe 30-50% of lambs born to an infected mother will test positive by 6 months of age. Other lambs in the herd frequently come up positive, also. Up to 17% of lambs from uninfected mothers have been found to have become infected in herds that include infected ewes.
Because lambs like to try and steal milk from other ewes, that may be a mode of transmission. OPP does not survive long in the environment and apparently needs to be transmitted via body fluids, similar to aids. Close winter housing seems to be a very high risk environment for transmission according to some studies. Needles are another route of transmission. One needle to one sheep is the rule for preventing transmission of all of these diseases. Extra needles are a minor cost compared to the economic damage these diseases do.
Research all three of these diseases and learn all you can so you’ll be prepared for the inevitable brush your herd will have with at least one of them. Quarantining and testing new sheep is one of the best preventions. Use the internet. There is a host of information available. New vaccines are being developed so it pays to keep up with the news.
The OPP Society website is a marvelous resource for breeders looking for solutions to managing infection in their herd. They recommend testing all animals and separating them into two herds. Routine testing at least once a year of the negative herd will pick up any animals missed or that contract the disease later. New positives are moved to the positive herd. The negative herd should not have nose or fecal contact with the positive herd, ideally separated by at least 200 feet.
The positive herd is slowly reduced by removing animals as they would normally be shipped. Ewes with valuable bloodlines are bred by AI and their lambs are removed at birth and not given any chance to nurse or be licked by their mothers. The OPP society page has some very good articles on successfully rearing orphan lambs. Colostrum can be saved from the negative herd or the lambs grafted to a negative ewe. Visit www.oppsociety.org
It’s recommended to continue testing all animals for two years after all known positives are off the property. Testing ewes after they give birth will give lots of false negatives because a majority of the ewes antibodies go into her milk and won’t be found in the blood. Testing lambs prior to 6 months, especially if they have nursed from a positive ewe, will give false positives because they have ingested their mother’s antibodies but may not have contracted disease themselves. Johnes antibodies frequently do not show up in the blood until an animal is 2 years old, even if it is present in the gut and shedding contagion in the feces.
One of the barriers to having more flocks test and eliminate these diseases is the cost of testing. This is not a huge problem if you have a small flock and collect your own samples (draw blood for all three diseases or collect feces for Johnes). Ideally, anyone starting up would test while their flock was still small and test any incoming animals. But there are many large flocks that cannot afford to test due to the number of sheep involved.
It would be a huge advantage to the whole industry if we could work with the USDA and the states to subsidize testing and start making progress eliminating these diseases. Many states are already trying to help with this and commonly, if the tests you need are available from you own state vet labs, the cost will be subsidized for you as a resident.
Our industry is small compared to the cattle industry and we need to make ourselves heard on this issue. Discuss your situation with your vet and call your state labs to find out what is possible.
It is much less expensive if you learn to draw the blood samples yourself. In most cases the serum is all that is required by the testing lab. You do not need to pay your vet to spin the blood to separate it.
Simply set the tubes of blood in a warm place over night and then use a clean hypodermic needle and syringe to carefully draw the clear serum off the coagulated blood cells and transfer it to a clean red top tube. Be careful not to cross contaminate your samples. Package them carefully, use an icepack in warm weather, and mail them overnight to the lab.
Purdue has an excellent web article demonstrating how to locate a sheep’s jugular vein and draw blood at www.ces.purdue.edu
Don’t bring a problem you don’t have into your herd. Don’t buy from herds that don’t test. Quarantine and test any animals you have any reason to be unsure of. Test your own herd regularly so you can catch any accidental introductions before they spread. Remember that shows, your neighbor’s flock, or other contact with the sheep community can bring infection into your flock unexpectedly. CL is also carried by goats. Johnes disease is spread by cow manure and cows milk.
Reputable breeders know that testing helps sell sheep. Good records allow you to market your sheep as disease free. Its good ethics, it’s good for our breed, and it’s good for your business.